GENEVA — Researchers at the University of Geneva have found that the speed at which the virus can spread and the complexity of its genetic makeup are the main reasons why it’s difficult to stop it.
In a study published Monday in the journal Science Translational Medicine, they say the virus has evolved over millions of years and that this has been the main reason why the spread of the disease is not being prevented.
“The more you look at it, the more you see that it’s not going to be solved,” said Dr. Martin Dittmar, a professor at the department of microbiology and immunology at the university and lead author of the study.
Dittmar and his team looked at the genomes of more than 3,000 strains of the coronavirus.
They looked at how they changed as the virus evolved and compared this to a genetic database of human genomes from more than 40,000 people who had been diagnosed with the disease.
Among the findings: The virus’s genetic make-up has evolved rapidly, but it’s still too complex for a virus to be effectively controlled by vaccines.
The virus also spreads faster in water than in air, which makes it more difficult to contain.
The virus has an enormous range of possible genes and mutations, and this means that it can evolve so quickly that its genetic make up changes.
It’s also difficult to make a vaccine because the virus is so complex and it takes a lot of time and money to produce a vaccine.
The scientists found that while some viruses evolve faster than others, the ones that are more complex are also more difficult for scientists to predict.
This is important because we know that many of the diseases we have in the world are caused by viral infections.
In the last decade, there has been an increase in pandemic-related infections, and they have all been caused by viruses.
Dittmars research showed that while the virus that causes dengue and other infections has a long sequence of mutations, the virus in humans can be rapidly evolving.
“We found that some viruses, such as the one that causes chikungunya, are very complicated to control, so they have to evolve faster,” Dittmer said.
“Other viruses, like the one responsible for dengua, are not that complicated.
And it is very easy for viruses to evolve and spread.”
For the new study, Dittsmar and two colleagues compared the genomes for more than 1,200 strains of coronaviruses.
Using the genetic data from these strains, they were able to map the virus genome and map the different types of mutations it had, which were the key factors in making the virus more difficult or less difficult to control.
The team found that all of the strains of virus were more complex than the ones they had previously studied.
The more complex the virus, the longer it took to evolve.
Researchers also found that mutations that cause a virus’s ability to spread were also the main factors.
As for the complexity, it can be as simple as changing the way the virus moves through a population.
For instance, one mutation that causes a virus that spreads quickly, called a caspase, is very common, so the researchers could have predicted this mutation by looking at a genetic dataset from thousands of people with the virus.
But when they looked at people with different mutations, they found that different mutations were associated with different diseases, including coronaviral encephalitis, which causes fever and muscle weakness.
There are different forms of the caspases, and scientists are trying to find ways to target the mutations that allow the virus to spread.
The researchers say it could be possible to create a vaccine that targets the caskets of a virus and that could then stop the virus from spreading further.
Although it’s clear that the new coronaviri is not as complex as previously thought, there is still a lot that scientists need to learn about the virus and its capabilities to infect and kill people.
Dittmares research was funded by the European Union.